Oral Tren vs Injectable tren
anyone used both? what were effects?
and generally speaking post here all info related to using both products.
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I never injected the Injectable tren. But i ran Oral Tren 2 times with amazing results.
Postive 2 tabs a day is all that is needed 3 at most. I used only 2 a day, 2 weeks at a time.
Positive effects- muscle results in less then a week usually you can tell your on it right away, but notice the results in 5 to 6 days. You get leaner fast. good strentgh gains aswell.
Long nights some times hard to sleep
thats all i can think of. I didnt get no bad sides but also you get bad nightmares, they can be scary.
ive never ran oral tren before, ive always heard it is the most liver toxic of all the orals. but i have ran injectible(acetate).
great strength gains
lean, hard look of a god
sweat 24/7, even in a 68 degree a/c controlled room
loss of endurance
oily, acne prone skin; the collars of all my white shirts turned yellow
this cycle was run at:
prop 100 m,w,f
tren 100 m,w,f
and cyp at 250/wk, was worried my dick wouldnt work at a 1:1 test:tren ratio
I belive the chemical name for Oral Trenbolone is Metribolone.
Pharmaceutical Name: Methyltrienolone
Chemical structure: 17-methylestra-4,9,11-trien-3-one,17b-ol
Molecular weight of base: 284.3974
Effective dose: 5-15 mg / day
Average Street-price: Only available for research purposes.
Available Doses: None
Brands & Products: Originally produced by Negma, but never approved for production.
Methyltrienolone is structurally similar to trenbolone (Parabolan/Finaplix), a well-liked and powerful androgen that does not aromatize to estrogen. The difference is the attachment of a 17-alpha-methyl group for oral activity. So one could refer to methyltrienolone as oral trenbolone. It was first explored quite some time ago by Negma in France, the same company that marketed Parabolan (trenbolone). But the drug was never approved by the French government and was hence never produced. The reason was extreme hepatoxicity. Bill Roberts, the biochemist, once commented that taking methyltrienolone made taking insane doses of anadrol and Halotestin together look mild on the liver. While I was unable to find anything in the literature that describes the extent of the liver toxicity, it's a generally accepted fact. That's also why, to the dissapointment of many, you will never find a commercially marketed methyltrienolone product. Its only sold in bulk to labs and universities for research studies involving androgens.
Mainly because (and those who wish it was available will wish so even more now) its such a potent androgen. There is some conflicting information in that regard however. Organic chemist Patrick Arnold, head of LPJ research, once stated that methyltrienolone was the most powerful steroid ever, and that statement has been blown out of proportion and taken on a life of its own. While androgenically a very potent steroid, methyltrienolone is still basically trenbolone with a 17-alpha-methyl group. A group that has the tendency to actually reduce the androgenic potency. So it may actually be somewhat milder than trenbolone, on the contrary to what many pseudo steroid guru's are now claiming after reading Pat Arnold's statement. I can't find any other documented effects of the 17-alpha-alkylation influencing androgen binding in a positive way. It's a potent androgen, with more binding than even DHT, but the study that claims that is mild at the very best about quantifications, whereas people have used the term 1000 times more powerful than testosterone, which is surely exaggerated.
What is interesting is that it seems to show nearly no binding for sex-hormone binding proteins, which makes it a popular choice in androgen receptor studies, since it will demonstrate equal binding in all tissues regardless of the presence and amount of these proteins. No doubt this plays a role in its supposed binding capacity. In this instance the 17-alpha-alkylation may have played a key role, since it has been demonstrated a multitude of times that 17-alpha-methyl groups decrease the binding for sex-hormone binding proteins as well as most other structures, and due to its triple double bond, trenbolone really didn't bind well to these to begin with.
One of the findings made in clinical tests with methyltrienolone was the discovery of high amounts of the DHT-deactivating enzyme 3alpha-hydroxysteroid dehydrogenase in muscle tissue. Once again proof that God meant to keep us humans weak. Hurray for science. Follow-up studies then went on to show that DHT nonetheless showed similar binding in the prostate, and showing little or no presence of the deactivating enzyme. So God would rather have us all die of prostate cancer than gain a few ounces of muscle. It's a comforting thought, no?
What methyltrienolone, despite its amazing capacity, still doesn't overcome are the basic problems with any 19Nor compound. First of all its effects on libido. Methyltrienolone still seems to affect our sex drive in such a potent manner that the dreaded Deca Dick (temporary impotence) is a very real threat. Another is that it still binds almost equipotently to the progesterone receptor. The latter would be of little concern as long as no circulating estrogen is present since methyltrienolone does not aromatize, but could cause problems such as aggravating water retention and gyno (growth of breast tissue in men) if combined with an aromatizing androgen or an estrogen.
While many may wish that an incredibly strong androgenic, non-aromatizing compound as this was available for daily use, its not. And if the indications are true, its probably best. I've warned many people for the toxicity of fluoxymesterone, and everything points to it that methyltrienolone makes fluoxymesterone look like Tums tablets in terms of liver toxicity.
Stacking and Use:
Obviously this section is mostly useless, as any who would use, let alone stack methyltrienolone for any decent period of time, wouldn't really be around long enough to tell us how well it worked. Ideally one would use it alone, while dieting or for the purpose of gaining lean mass. The androgenic potency is slightly higher than that of trenbolone, so the risk for aggravated hair loss, acne, prostate hypertrophy and deepening of voice is not only realistic, but almost likely. If one were to use it, you would probably have to use every trick in the book to protect your liver and stay alive: Alpha Lipoic Acid, Milk thistle, dessicated liver and Vitamin B6. The blood pressure raise would not be mild either. So something to lower blood pressure is advised as well.
Last edited by Rex; 02-15-2011 at 05:16 AM.
I have been reading about Tren and have found wide spread confusion about it mainly because when one writes "Tren," s/he could be referring to at least 5 different compounds:
1. Trenbolone Acetate - injectable version (Finaject and Finajet)
2. Trenbolone Acetate - pellet form known as Finaplix
3. Trenbolone Cyclohexylmethylcarbonate - (called Parabolan by Bill Roberts)
4. Trenbolone Hexahydrobencylcarbonate - (called Parabolan on many message boards)
5. Trenbolone Enanthate
The many names of Tren have confused many people. I've seen Veterans and Moderators and Members of several boards equate one trenbolone compound with another, not knowing that one is actually not equivalent to the other. One common mistake I see is calling Trenbolone Hexahydrobencylcarbonate "Tren Enanthate." They are similar, but not the same (see below). I also have seen people refer to Trenbolone Acetate as Parabolan--wrong!
So, let's clear this up.
The following is a brief summary of the main differences of each that I have created in order to clear up my own confusion on Trenbolone and hopefully help others here in the process. It is not meant to provide a detailed description of Tren activity in the body.
1. Trenbolone Acetate--injectable version (Finaject and Finajet)
This is correctly referred to as "Fina." Finaject is the acetate form of trenbolone. It was produced in a short acting ester (acetate), so its effect lasts only a short time and frequent administration is necessary. Finaject was an injectable steroid of veterinary medicine, which was extremely popular in bodybuilding and powerlifting during the 1980's.
The injectable Trenbolone Acetate called Finaject is no longer produced.
(Refer to the end of this post for a discussion of Esters)
2. Trenbolone Acetate--pellet form Finaplix
Finaplix was a veterinary cattle implant, which contained the potent androgenic steroid Trenbolone Acetate. Once Finaject and Finajet were no longer manufactured, bodybuilders began using Finaplix to make topical or injectable versions of Trenbolone Acetate.
Today, cattle implants have become designer products with varied doses and combinations of estrogenic and/or androgenic (trenbolone) agents. So, the process of converting cattle implants to useful versions of trenbolone acetate has become more difficult since one must separate the trenbolone from the other additives present in the cattle implants before using it.
3. Trenbolone Cyclohexylmethylcarbonate (called Parabolan by Bill Roberts)
Parabolan contains a much different ester than Finaject and Finajet, called Trenbolone Cyclohexylmethylcarbonate. This ester extends the activity of trenbolone for more than two weeks, a more suitable design for human use.
The amount of trenbolone in 76 mg of Trenbolone Cyclohexylmethylcarbonate is equivalent to the amount of trenbolone in only 58 mg of Trenbolone Acetate. The acetate is a little more potent, more effective per milligram, because the acetate ester is lighter than the cyclohexylmethylcarbonate ester; therefore a higher percentage of the weight of Trenbolone Acetate is trenbolone. A similar comparison also can be made with the other long lasting esters of trenbolone: enanthate and Hexahydrobencylcarbonate.
The muscle building properties of Trenbolone Cyclohexylmethylcarbonate are the same as Trenbolone Acetate (Finaject or Finajet) except for the longer half-life.
Although it is very similar, this compound is NOT the same as Trenbolone Enanthate. The only difference in these compounds is the esters (see ester definitions below), which all act almost identically (long lasting esters).
4. Trenbolone Hexahydrobencylcarbonate (called Parabolan on many message boards)
Trenbolone Hexahydrobencylcarbonate and Trenbolone Cyclohexylmethylcarbonate are exactly the same substances. Hexahydrobencylcarbonate ester is just another name for cyclohexylmethylcarbonate ester.
5. Trenbolone Enanthate
Although it is very similar, this compound is NOT the same as Trenbolone Cyclohexylmethylcarbonate (Trenbolone Hexahydrobencylcarbonate). The only difference in these compounds is the esters (see ester definitions below).
THE DIFFERENCE BETWEEN THE ESTERS
The most important difference between the esters is whether it is a short acting ester or a long lasting ester. The next most important difference is the weight of the ester. As mentioned under the Trenbolone Cyclohexylmethylcarbonate section (above), the relative potency of each ester of trenbolone is partially dependent on the weight of its ester.
The main difference between different esters is simply the number of carbon atoms in the ester. Propionate has three carbons, acetate has two, isobutyrate has four, enanthate has seven, cypionate has eight, and deaconate has ten. More unusual esters, such as cyclohexylmethylcarbonate (used in Parabolan) has eight carbons and one more oxygen than the above esters making it the heaviest.
Therefore, the esters of trenbolone in order of potency when compared milligram to milligram (from most potent to least):
1. Tren Acetate
2. Tren Enanthate
3. Tren Cyclohexylmethylcarbonate (Tren Hexahydrobenzylcarbonate)
The differences in potency caused by the esters are negligible. So, you should base your choice of Trenbolone on how frequently you plan to inject, how much you trust your supplier, and how much you trust the brand of tren you purchase.
If you are concerned about the possible side effects of Trenbolone, and don't mind frequent injections, then consider using Trenbolone Acetate. If bad side effects manifest, Tren Acetate will quickly leave your body after the last injection due to the short acting ester (acetate); and your body will be able to begin to recover quickly. On the contrary, your recovery from bad side effects won't begin until 2 weeks after the last injection of a long lasting ester of tren because a long lasting ester of tren will stay active in your body for more than two weeks after your last injection--continuing to contribute to the bad side effects.
Last edited by Rex; 02-15-2011 at 05:24 AM.